sempre sul tema scie chimiche,
Qua si da indirettamente notizia del fatto che i militari spruzzino
aereosol di solfato di bario nell'atmosfera
sul sito governativo statunitense pubmed
http://www.ncbi.nlm.nih.gov/pubmed?cmd=Retrieve&db=pubmed&dopt=AbstractPlus&list_uids=15082100&query_hl=2
Chronic barium intoxication disrupts sulphated proteoglycan synthesis: a
hypothesis for the origins of multiple sclerosis.
Purdey M.
Source
High Barn Farm, Elworthy, Taunton, Somerset TA43PX, UK.
tsepurdey@???
Abstract
High level contamination by natural and industrial sources of the alkali
earth metal, barium (Ba) has been identified in the
ecosystems/workplaces that are associated with high incidence clustering
of multiple sclerosis (MS) and other neurodegenerative diseases such as
the transmissible spongiform encephalopathies (TSEs) and amyotrophic
lateral sclerosis (ALS). Analyses of ecosystems supporting the most
renowned MS clusters in Saskatchewan, Sardinia, Massachusetts, Colorado,
Guam, NE Scotland demonstrated consistently elevated levels of Ba in
soils (mean: 1428 ppm) and vegetation (mean: 74 ppm) in relation to mean
levels of 345 and 19 ppm recorded in MS-free regions adjoining. The high
levels of Ba stemmed from local quarrying for Ba ores and/or use of Ba
in paper/foundry/welding/textile/oil and gas well related industries, as
well as from the use of Ba as an atmospheric aerosol spray for
enhancing/refracting the signalling of radio/radar waves along military
jet flight paths, missile test ranges, etc. It is proposed that chronic
contamination of the biosystem with the reactive types of Ba salts can
initiate the pathogenesis of MS; due to the conjugation of Ba with free
sulphate, which subsequently deprives the endogenous sulphated
proteoglycan molecules (heparan sulfates) of their sulphate co partner,
thereby disrupting synthesis of S-proteoglycans and their crucial role
in the fibroblast growth factor (FGF) signalling which induces
oligodendrocyte progenitors to maintain the growth and structural
integrity of the myelin sheath. Loss of S-proteoglycan activity explains
other key facets of MS pathogenesis; such as the aggregation of
platelets and the proliferation of superoxide generated oxidative
stress. Ba intoxications disturb the sodium-potassium ion pump--another
key feature of the MS profile. The co-clustering of various
neurodegenerative diseases in these Ba-contaminated ecosystems suggests
that the pathogenesis of all of these diseases could pivot upon a common
disruption of the sulphated proteoglycan-growth factor mediated
signalling systems. Individual genetics dictates which specific disease
emerges at the end of the day.
PMID: 15082100 [PubMed - indexed for MEDLINE]
